Captain Oblivious wrote:
Treads for wrote:Everyone is physiologically different. One rider could have a natural heamocrite of 42 and the other could have a natural haemocrite of 45. If they both perform at the same level when not doping, if they both take EPO and get their haemocrite up to 50, the rider with the natural 42% reading will destroy the rider with the natural 45% reading.
And by the way, EPO is already used to treat cancer patients after chemo to help create red blood cell growth after radiation therapy.
Prove your first statement.
And thanks tips, for your second one.
This is not a subject with peer-reviewed studies on it for obvious reasons. It is what I have learned from years as a cyclist. If a high performing athlete came in to the team and had a low natural haemocrite reading, the team doctor would be rubbing his hands together.
A low natural level means you can take more EPO and still be in the safe zone in tests. The half life of EPO microdosing is about 4 hours for a synthetic test which makes the chances of being caught that way very small. An increase of 42 to 50 is a bigger increase than 45 to 50. The first rider gets 8% more RBCs after EPO while the second only gets 5%.